This does not address my initial question...
And this ignores the rest of that section of the post.
I asked for documented proof -...
I have a question for you. Given that we know and have established that lab rodent colonies have parasites, at least some of which are also parasites in wild snakes, do you think there is something somehow magical about their parasites and captive bred snakes that somehow prevents transmission? As that is what your position effectively requires you to believe in order to demand documentation in this manner. I should not need it, as it should be plainly evident.

Another question: Why are the really nice high end frozen mice irradiated if people are not paying a premium to prevent parasites?

Of course, that is now irrelevant, as I have found a rather large survey of cryptosporidium in zoo collections. Guess what I have found? C. parvum mouse genotype, found in Elaphe obsoleta, Python regius, Boa constrictor, Lampropeltis geluta, Elaphe guttata, Corallus caninus, L. triangulum, L. calligaster, and Elaphe vulpina... A well as a lot of non-snakes.

The reference for this is

Xiao, L. Et al. 2004. Genetic Diversity of Cryptosporidium spp. in Captive Reptiles. Applied and Environmental Microbiology, 70(2):891-899.

Indeed. And speculation is not fact - it's a guess.
No. Lay person speculation is a guess. I am not a lay person and am speculating from a specialized knowledge base. I am synthesizing information from across a field of study and taking it to its next logical step. That is not a guess. The only place where I am in any way making a jump, is because I do not know the exact parasite load of the average lab mouse, and thus cannot tell you what parasites can use a reptile as a vector. Everything else in that little paragraph is stuff I can teach to a freshman biology class and not have any controversy on unless a creationist makes it into the University of Texas Board of Regents. Thankfully Gov Perry( A pox be upon him) has no say in such matters.

I love how in your response you completely ignore the entire chain of logic I use to reach that conclusion. Did you read and understand it?

Yes, see Le Ann's post for more information.
A lay person's anecdotes are not evidence. Plus it is dealing with 1 out of many species, under non-controlled conditions, in a different clade than H. cinerea (Pacific treefrogs are within Pseudacris now)

"Affecting" survival is not the same as killing.
On a population level, yeah, it is.

but you have no evidence whatsoever that parasites all by their lonesome, kill wild snakes
very little will kill anything "all on its lonesome" and that is never what I claimed. I grow tired of you cherry picking snippets out of context and generally misrepresenting my arguments. If you want to know why I insult you, that is why.

Here is the original statement.

Snakes in the wild die from their parasite loads all the time. They die because they get too cold, and because they dont get enough food, or they catch another pathogen. The wild is not some magical clean place where everything is Happy Smiley Fun Times. You dont swim in a lot of the water that garter snakes live in because you will develop a lethal protazoan infection. The snakes deal with that every day.

Nothing dies from one cause typically, unless their predators use a random predation mode (like filter feeding) or they catch something with a near 100% mortality rate (ebola in humans for example... or a heavy load of chinese liver flukes). They die because they are immune suppressed due to cold and cant fight off the parasites, or because they have a bad food year and their energy intake goes below their parasite-enhanced maintenance threshold and they deplete their fat reserves, at which point they either starve or get picked off by a predator before starving. Either way the parasite load is one of many proximate and ultimate causes of mortality, and I did not claim otherwise.

This depends on the parasites of course. Some parasites will of course kill their hosts... certain flukes for example, as will Plasmodium. However that you wont detect in the wild because the animal decays and dies before you find it. The only way to detect death from parasitism in this manner is through... *gasp* correlating parasite load with recapture probability. Just like the study I cited.

especially with the frequency of that "all the time" nonsense you've stated.
The per annum mortality rate for juvenile T. elegans along Eagle Lake California alone is 80-90% (I would need to dig up the papers again to get the exact number), a portion of that is directly and indirectly due to parasites, and we are talking about thousands of little snakes per year.

T. sirtalis in Manitoba have high mortality rates from year to year as well due to "poor body condition" during hibernation. The guys that do the work never performed a necropsy on the dead ones, however poor body condition is contributed to by parasite loads.

What? Do you think that parasites just magically kill the animals? Most parasites dont actually create some sort of accute illness that by itself will kill a snake. The helminth parasites at least reduce energy and nutrient intake from food and kill indirectly via starvation. Blood parasites are a different matter, but I know little about them other than what I described above.

Maybe you should stick to subject matter that you know (whatever that may be).
Here is my area of study:

Foraging strategies in Natricine snakes, and the evolution of novel predator recognition and anti-predator strategies in frogs toward natricine snakes. Anti-predator strategies include in this case chemical defenses, behavioral defenses, and life history shifts. It also requires me to know about most of the things which are ancillary to this. Enegy budgets, sexual selection, costs of reproduction etc. I dont work with parasites, but host-parasite interactions are a subset of predator-prey interactions and they rely on the same concepts. The scale of application is just smaller.

Sounds like you're making a personal insult.
Actually no. It is a personal and public service. I just have a caustic personality, you should see me on a board with looser rules of engagement. I would also note you are committing a Style over Substance Fallacy. I am not a caustic jerk as a substitute for an argument. If you will notice, I refuted your argument (and have expanded on this from there) and then I followed by being a caustic jerk. The two are entirely different things.

Instructive example

Person 1: 2+2=5
Person 2: No. You are wrong because your (insert your mom joke here)

That is what is called an ad hominem attack, which is what you accuse me of.

What I actually do is this

Person 1: 2+2=47
Person 2: (Proof involving counting of apples and showing the other person a numberline), oh and by the way (insert your mom joke here)

You will notice how the insult is secondary to the argument. In my case, they are typically the result of frustration when someone either does not read, does not comprehend, or deliberately misrepresents my posts. All three of which are things you have demonstrated.

Yes, but if you can't prove mice have parasites that are transferrable to captive snakes (especially lab mice) why not simply feed your snakes rodents, instead of the hassle of raising frogs (if it's even possible) and STILL having the possibility that the captive-raised frogs will have parasites that are transferrable to the snake.
Because I have now established that at least cryptosporidium (and probably a lot of other stuff, like tapeworms), is transfered. A snake which does not naturally prey upon rodents will have no defenses against a parasite that uses the rodent as an obligate or facultative intermediate host, save to "hope" it gets enough food (in the case of an intestinal parasite) to get what it needs after the parasite takes its cut, or that the parasite load is low enough to not cause problems. A snake which preys upon rodents will have a variety of defenses against such parasites (the parasite will determine what those are, from chemical to cellular, to energy allocation shifts, and thermoregulatory changes)

In the same way, a snake which preys upon fish and frogs will have more defenses against the parasites that use them as hosts than those which do not.

Not all parasites are equal. You also need to consider per-parasite risk

As for me. Did my undergraduate work at ASU, now I am at the University of Texas, Arlington.